Methyltestosterone steroidology

Endogenous androgens are responsible for the normal growth and development of the male sex organs and for maintenance of secondary sex characteristics. These effects include the growth and maturation of prostate , seminal vesicles , penis , and scrotum ; the development of male hair distribution, such as beard , pubic, chest and axillary hair, laryngeal enlargement, vocal cord thickening, alterations in body musculature, and fat distribution. Drugs in this class also cause retention of nitrogen , sodium, potassium , phosphorus , and decreased urinary excretion of calcium. Androgens have been reported to increase protein anabolism and decrease protein catabolism . Nitrogen balance is improved only when there is sufficient intake of calories and protein. Androgens are responsible for the growth spurt of adolescence and for the eventual termination of linear growth centers. In children, exogenous androgens accelerate linear growth rates, but may cause a disproportionate advancement in bone maturation. Use over long periods may result in fusion of the epiphyseal growth centers and termination of growth process. Androgens have been reported to stimulate the production of red blood cells by enhancing the production of erythropoietic stimulating factor.

Methyltestosterone is the INN , USAN , USP , BAN , and JAN of the drug and its generic name in English and Japanese , while méthyltestostérone is its DCF and French name and metiltestosterone is its DCIT and Italian name. [3] [4] [5] The generic name of the drug is methyltestosterone in Latin , methyltestosteron in German , and metiltestosterona in Spanish . [3] [4] [5] Methyltestosterone is also known by its former developmental code name NSC-9701 . [5]

Methyltestosterone steroidology

methyltestosterone steroidology

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